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Frontiers | A20/Tumor Necrosis Factor α-Induced Protein 3 in Immune Cells Controls Development of Autoinflammation and Autoimmunity: Lessons from Mouse Models

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管理番号 新品 :485437630
中古 :485437630-1
メーカー a6048 発売日 2025-04-04 00:27 定価 6000円
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Frontiers | A20/Tumor Necrosis Factor α-Induced Protein 3 in Immune Cells Controls Development of Autoinflammation and Autoimmunity: Lessons from Mouse Models

Frontiers | A20/Tumor Necrosis Factor α-Induced Protein 3 in Immune Cells  Controls Development of Autoinflammation and Autoimmunity: Lessons from  Mouse ModelsFrontiers | A20/Tumor Necrosis Factor α-Induced Protein 3 in Immune Cells Controls Development of Autoinflammation and Autoimmunity: Lessons from Mouse Models,Correction of a homoplasmic mitochondrial tRNA mutation in patient-derived  iPSCs via a mitochondrial base editor | Communications BiologyCorrection of a homoplasmic mitochondrial tRNA mutation in patient-derived iPSCs via a mitochondrial base editor | Communications Biology,OCT4/SOX2‐independent Nanog autorepression modulates heterogeneous Nanog  gene expression in mouse ES cells | The EMBO JournalOCT4/SOX2‐independent Nanog autorepression modulates heterogeneous Nanog gene expression in mouse ES cells | The EMBO Journal,MAB21L1 promotes survival of lens epithelial cells through control of  αB-crystallin and ATR/CHK1/p53 pathway | AgingMAB21L1 promotes survival of lens epithelial cells through control of αB-crystallin and ATR/CHK1/p53 pathway | Aging,MAB21L1 promotes survival of lens epithelial cells through control of  αB-crystallin and ATR/CHK1/p53 pathway | AgingMAB21L1 promotes survival of lens epithelial cells through control of αB-crystallin and ATR/CHK1/p53 pathway | Aging,

 

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